for Journals by Title or ISSN
for Articles by Keywords
help
  Subjects -> BIOLOGY (Total: 2541 journals)
    - BIOCHEMISTRY (188 journals)
    - BIOENGINEERING (55 journals)
    - BIOLOGY (1298 journals)
    - BIOPHYSICS (41 journals)
    - BIOTECHNOLOGY (144 journals)
    - BOTANY (188 journals)
    - CYTOLOGY AND HISTOLOGY (25 journals)
    - ENTOMOLOGY (50 journals)
    - GENETICS (136 journals)
    - MICROBIOLOGY (200 journals)
    - MICROSCOPY (9 journals)
    - ORNITHOLOGY (23 journals)
    - PHYSIOLOGY (66 journals)
    - ZOOLOGY (118 journals)

BIOLOGY (1298 journals)            First | 6 7 8 9 10 11 12 13 | Last

Micron     Hybrid Journal  
Mitochondrial DNA     Hybrid Journal   (3 followers)
Mitochondrion     Hybrid Journal   (3 followers)
Modelling and Simulation in Engineering     Open Access   (4 followers)
Modelling and Simulation in Materials Science and Engineering     Partially Free   (7 followers)
Molecular & Cellular Proteomics     Full-text available via subscription   (7 followers)
Molecular & Cellular Toxicology     Hybrid Journal   (2 followers)
Molecular and Biochemical Parasitology     Hybrid Journal   (1 follower)
Molecular and Cellular Biochemistry     Hybrid Journal   (4 followers)
Molecular and Cellular Biology     Full-text available via subscription   (16 followers)
Molecular Based Mathematical Biology     Open Access  
Molecular Biology     Hybrid Journal   (8 followers)
Molecular Biology and Evolution     Hybrid Journal   (94 followers)
Molecular Biology International     Open Access   (3 followers)
Molecular Biology of the Cell     Partially Free   (14 followers)
Molecular Biology Reports     Hybrid Journal   (4 followers)
Molecular Brain     Open Access   (2 followers)
Molecular Breeding     Hybrid Journal   (6 followers)
Molecular Cell     Full-text available via subscription   (27 followers)
Molecular Ecology     Hybrid Journal   (14 followers)
Molecular Ecology Resources     Hybrid Journal   (8 followers)
Molecular Genetics and Metabolism     Hybrid Journal   (6 followers)
Molecular Immunology     Hybrid Journal   (11 followers)
Molecular Membrane Biology     Hybrid Journal   (1 follower)
Molecular Neurobiology     Hybrid Journal   (3 followers)
Molecular Pain     Open Access  
Molecular Plant-Microbe Interactions     Partially Free   (5 followers)
Molecular Reproduction & Development     Hybrid Journal   (6 followers)
Molecular Therapy - Nucleic Acids     Open Access  
Molecules and Cells     Hybrid Journal   (1 follower)
Momona Ethiopian Journal of Science     Open Access  
Monoclonal Antibodies in Immunodiagnosis and Immunotherapy     Hybrid Journal  
Monographs of the Western North American Naturalist     Full-text available via subscription   (1 follower)
Moscow University Physics Bulletin     Hybrid Journal   (1 follower)
Movement Ecology     Open Access   (3 followers)
Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis     Hybrid Journal   (2 followers)
Mutation Research/Genetic Toxicology and Environmental Mutagenesis     Hybrid Journal   (7 followers)
Mutation Research/Reviews in Mutation Research     Hybrid Journal   (4 followers)
Mycological Progress     Hybrid Journal   (1 follower)
Mycopathologia     Hybrid Journal   (1 follower)
Mycorrhiza     Hybrid Journal   (5 followers)
Mycoscience     Hybrid Journal   (1 follower)
Mycotoxin Research     Hybrid Journal   (4 followers)
NAMMCO Scientific Publications     Open Access  
Nano Reviews     Open Access   (15 followers)
Nano Today     Hybrid Journal   (10 followers)
Nanoscale Research Letters     Open Access   (4 followers)
Natural Hazards     Hybrid Journal   (97 followers)
Natural History     Full-text available via subscription   (4 followers)
Natural Product Research: Formerly Natural Product Letters     Hybrid Journal   (1 follower)
Natural Science     Open Access   (9 followers)
Nature     Full-text available via subscription   (1874 followers)
Nature Cell Biology     Full-text available via subscription   (149 followers)
Nature China     Full-text available via subscription   (5 followers)
Nature Digest     Full-text available via subscription   (4 followers)
Nature Protocols     Full-text available via subscription   (33 followers)
Nature Reviews Molecular Cell Biology     Full-text available via subscription   (124 followers)
Nature Structural & Molecular Biology     Full-text available via subscription   (120 followers)
Naturwissenschaften     Hybrid Journal   (4 followers)
Nematology     Full-text available via subscription   (2 followers)
Nematropica     Open Access  
NeoBiota     Open Access   (2 followers)
Neotropical Ichthyology     Open Access  
Nepal Journal of Science and Technology     Open Access  
Neurobiology of Aging     Hybrid Journal   (3 followers)
Neurobiology of Disease     Hybrid Journal   (3 followers)
NeuroImmune Biology     Full-text available via subscription  
Neuroinformatics     Hybrid Journal   (2 followers)
Neuropeptides     Hybrid Journal   (2 followers)
Neuropsychobiology     Full-text available via subscription   (3 followers)
New Biotechnology     Hybrid Journal   (4 followers)
New Journal of Physics     Open Access   (7 followers)
New Phytologist     Hybrid Journal   (10 followers)
New Zealand Journal of Marine and Freshwater Research     Hybrid Journal   (4 followers)
Nigerian Journal of Basic and Applied Sciences     Open Access   (2 followers)
Nigerian Journal of Parasitology     Full-text available via subscription  
Nigerian Journal of Physiological Sciences     Open Access  
NJAS - Wageningen Journal of Life Sciences     Full-text available via subscription   (1 follower)
NMR in Biomedicine     Hybrid Journal   (1 follower)
Non-Genetic Inheritance     Open Access   (1 follower)
Nonlinear Biomedical Physics     Open Access  
Northern Territory Naturalist     Full-text available via subscription  
Northwest Science     Full-text available via subscription   (1 follower)
Nova Hedwigia     Full-text available via subscription   (2 followers)
Nuclear Medicine and Biology     Hybrid Journal   (1 follower)
Nucleic Acid Therapeutics     Hybrid Journal  
Nucleic Acids Research     Open Access   (24 followers)
Nucleic Acids Symposium Series     Hybrid Journal  
Nucleosides, Nucleotides and Nucleic Acids     Hybrid Journal   (1 follower)
Nucleus     Full-text available via subscription  
Nutrition & Metabolism     Open Access   (5 followers)
OA Bioinformatics     Open Access   (1 follower)
OA Stem Cells     Open Access  
Oceanological and Hydrobiological Studies     Hybrid Journal   (3 followers)
OMICS: A Journal of Integrative Biology     Hybrid Journal   (3 followers)
OnLine Journal of Biological Sciences     Open Access   (1 follower)
Open Biology     Open Access   (2 followers)
Open Journal of Apoptosis     Open Access  
Open Journal of Applied Biosensor     Open Access   (1 follower)
Open Journal of Biophysics     Open Access   (1 follower)

  First | 6 7 8 9 10 11 12 13 | Last

Biochemistry and Cell Biology    [10 followers]  Follow    
  Full-text available via subscription Subscription journal
     ISSN (Print) 0829-8211 - ISSN (Online) 1208-6002
     Published by NRC Research Press Homepage  [18 journals]   [SJR: 1.488]   [H-I: 65]
  • Expression and subcellular localization of RAGE in melanoma cells
    • Authors: Ioana Popa; Elena Ganea, Stefana M. Petrescu
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 127-136, e-First articles. The receptor for advanced glycation end products (RAGE) is involved in multiple stages of tumor development and malignization. To gain further knowledge on the RAGE role in tumor progression, we investigated the receptor expression profile and its subcellular localization in melanoma cells at different stages of malignancy. We found that RAGE clustered at membrane ruffles and leading edges, and at sites of cell-to-cell contact in primary melanoma cells (e.g., MelJuSo), in contrast with a more dispersed localization in metastatic cells (e.g., SK-Mel28). RAGE silencing by RNAi selectively inhibited migration of MelJuSo cells, whilst having no influence on SK-Mel28 cell migration, in a “wound healing” assay. Western blot detection of RAGE showed a more complex RAGE oligomerization in MelJuSo cells compared to melanocytes and SK-Mel28 cells. By competing the binding of antibodies with recombinant soluble RAGE, an oligomeric form running at approximately 200 kDa was detected, as it was the monomeric RAGE of 55–60 kDa. SDS-PAGE electrophoresis under reducing versus nonreducing conditions indicated that the oligomer of about 200 kDa is formed by disulfide bonds, but other interactions are likely to be important for RAGE multimerization in melanoma cells. Immunofluorescence microscopy revealed that treatment with two cholesterol-chelating drugs, nystatin and filipin, significantly affected RAGE localization in MelJuSo cells. SK-Mel28 cells showed a reduced RAGE glycosylation and association with cholesterol-rich membranes and also a considerable downregulation of the soluble forms. Our results indicate that RAGE isoform expression and subcellular localization could be important determinants for the regulation of its function in tumor progression.
      PubDate: Mon, 31 Mar 2014 12:13:11 GMT
       
  • Chicken biliary exosomes enhance CD4+T proliferation and inhibit ALV-J
           replication in liver
    • Authors: Yue Wang; Guihua Wang, Zhenzhen Wang, Huangge Zhang, Li Zhang, Ziqiang Cheng
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 145-151, e-First articles. Exosomes, which are small membrane vesicles of endocytic origin, carry lipids, RNA/miRNAs, and proteins and have immune modulatory functions. In this study, we isolated exosomes from the bile of specific pathogen-free chickens, 42–43 days of age, by using an ultracentrifugation and filtration method. The density of the exosomes, isolated by sucrose gradient fractionation, was between 1.13 and 1.19 g/mL. Electron microscopic observation of the liver showed that exosomes were present in the space of Disse and bile canaliculus. Chicken biliary exosomes displayed typical saucer-shaped, rounded morphology. Using liquid chromatography mass spectrum methodology, 196 proteins, including exosomal markers and several unique proteins, were identified and compared with mouse biliary exosomes. Noteworthy, CCCH type zinc finger antiviral protein was found on chicken biliary exosomes never described before. Furthermore, our data show that chicken biliary exosomes promote the proliferation of CD4+ and CD8+ T cells and monocytes from liver. In addition, chicken biliary exosomes significantly inhibit avian leukosis virus subgroup J, which is an oncogenic retrovirus, from replicating in the DF-1 cell line. These data indicate that chicken biliary exosomes possess the capacity to influence the immune responses of lymphocytes and inhibit avian leukosis virus subgroup J (ALV-J).
      PubDate: Fri, 14 Mar 2014 07:00:00 GMT
       
  • A new mechanism in the binding between Homer3 EVH1 domain and inositol
           1,4,5 trisphosphate receptor suppressor domain
    • Authors: He Wen; Hyuk Nam Kwon, Sunghyouk Park
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 1-9, e-First articles. The suppressor domain of inositol 1,4,5 trisphosphate receptor (IP3R) has critical roles in regulating the calcium channel by interacting with many binding partners. The residue 49–53 (PPKKF) of the suppressor domain was suggested to be a canonical Homer EVH1 domain binding site and is also the first a part of calmodulin (CaM) binding site. As CaM-binding of the suppressor domain has been shown to involve large-scale conformational changes, we studied the binding characteristics of the Homer EVH1-suppressor domain with NMR spectroscopy and biochemical pull-down assays for mutants. Our data show that the suppressor domain employs the PPKKF motif in a similar but subtly different way compared to previously characterized interactions, and that the suppressor domain does not undergo large-scale conformational changes. Chemical shift assignments of the Homer3 EVH1 domain found that a new set of residues, located at the opposite side of the previously reported binding site, is also involved in binding, which was confirmed by mutant binding assays. Further analysis suggests that F40 in the new binding sites may have a critical role as a conformational lock-switch in Homer-target binding. The proposed mechanism is implicated in the signaling network involving calcium channels.
      PubDate: Mon, 10 Mar 2014 07:00:00 GMT
       
  • Functional analysis of miR-101-3p and Rap1b involved in hepatitis B
           virus-related hepatocellular carcinoma pathogenesis
    • Authors: Yanrui Sheng; Shijia Ding, Ke Chen, Juan Chen, Sen Wang, Chengcheng Zou, Jingnan Zhang, Yiyi Cao, Ailong Huang, Hua Tang
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 152-162, e-First articles. MicroRNA-101(miR-101) has been shown to be down-regulated in hepatocellular carcinoma (HCC). The hepatitis B virus (HBV) is a major risk factor in the development and progression of HCC. However, the correlation between HBV and miR-101 has not yet been fully elucidated. In this study, we reported that HBV could repress miR-101-3p by inhibiting its promoter activity and identified the potential effects of miR-101-3p on some important biological properties of HCC cells by targeting Rap1b. Dual-luciferase reporter assays showed that HBV down-regulated miR-101-3p by inhibiting its promoter activity. Down-regulation of miR-101-3p promoted cell proliferation, migration, and reduced apoptosis, and resulted in up-regulation of Rap1b, while overexpression of miR-101-3p inhibited these processes. Moreover, overexpression of Rap1b was able to reverse the suppressed cell proliferation and migration mediated by miR-101-3p. Our data showed that HBV down-regulated miR-101-3p expression by inhibiting its promoter activity, which resulted in up-regulation of Rap1b, and down-regulation of miR-101-3p or up-regulation of Rap1b promoted proliferation and migration of HCC cells. This provides a new understanding of the mechanism of HBV-related HCC pathogenesis and the potential application of miR-101-3p in cancer therapy.
      PubDate: Mon, 10 Mar 2014 07:00:00 GMT
       
  • Hydrodynamic tail vein injection of SOCS3 eukaryotic expression vector in
           vivo promoted liver lipid metabolism and hepatocyte apoptosis in mouse
    • Authors: Zhenjiang Liu; Lu Gan, Xiaobo Yang, Zhenzhen Zhang, Chao Sun
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 119-125, e-First articles. Suppressor of cytokine signaling 3 (SOCS3), a signal transduction cytokine, is involved in lipid metabolism as well as in cell proliferation, differentiation, apoptosis, and so on. To explore the effects of SOCS3 on apoptosis and lipid metabolism in liver, we used a simple effective method named hydrodynamic tail vein injection to overexpress SOCS3. Then orbital blood was obtained for the assessment of blood lipid after injection. Lipid metabolism related genes were detected by Western blot after the determination of serum lipids. Meanwhile, liver cell apoptosis was observed by Hoechst and TUNEL staining and the expression of apoptosis related proteins Bax, Bcl-2, and Caspase3 were detected as well as the JAK2/STAT3 signaling pathway. In addition, we also demonstrated the effect of SOCS3 in prime hepatocyte by overexpression or interference of SOCS3 along with SD1008, which is a specific inhibitor of the JAK2/STAT3 signaling pathway. Taken together, all the results indicated that SOCS3 promoted lipid synthesis in mice liver and promoted hepatocyte apoptosis by inhibiting the activation of the JAK2/STAT3 signaling pathway, however the detailed regulation mechanism had not yet been fully understood and needs further study.
      PubDate: Wed, 19 Feb 2014 08:00:00 GMT
       
  • Different redox sensitivity of endoplasmic reticulum associated
           degradation clients suggests a novel role for disulphide bonds in
           secretory proteins
    • Authors: Iria Medraño-Fernandez; Claudio Fagioli, Alexandre Mezghrani, Mieko Otsu, Roberto Sitia
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 113-118, e-First articles. To maintain proteostasis in the endoplasmic reticulum (ER), terminally misfolded secretory proteins must be recognized, partially unfolded, and dislocated to the cytosol for proteasomal destruction, in a complex process called ER-associated degradation (ERAD). Dislocation implies reduction of inter-chain disulphide bonds. When in its reduced form, protein disulphide isomerase (PDI) can act not only as a reductase but also as an unfoldase, preparing substrates for dislocation. PDI oxidation by Ero1 favours substrate release and transport across the ER membrane. Here we addressed the redox dependency of ERAD and found that DTT stimulates the dislocation of proteins with DTT-resistant disulphide bonds (i.e., orphan Ig-μ chains) but stabilizes a ribophorin mutant (Ri332) devoid of them. DTT promotes the association of Ri332, but not of Ig-µ, with PDI. This discrepancy may suggest that disulphide bonds in cargo proteins can be utilized to oxidize PDI, hence facilitating substrate detachment and degradation also in the absence of Ero1. Accordingly, Ero1 silencing retards Ri332 degradation, but has little if any effect on Ig-µ. Thus, some disulphides can increase the stability and simultaneously favour quality control of secretory proteins.
      PubDate: Tue, 04 Feb 2014 08:00:00 GMT
       
  • Proposed cytotoxic mechanisms of the saffron carotenoids crocin and
           crocetin on cancer cell lines
    • Authors: Se Hyeuk Kim; Jung Min Lee, Sun Chang Kim, Chan Bae Park, Pyung Cheon Lee
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 105-111, e-First articles. We investigated the cytotoxic activities of crocin and crocetin, 2 major carotenoids isolated from the stigma of Crocus sativus (saffron), on 5 human cancer cell lines and proposed their possible anticancer mechanisms. Crocetin, a glycosylated carotenoid, showed approximately 5- to 18-fold higher cytotoxicity than crocin, a carboxylic carotenoid (IC50 of 0.16–0.61 mmol/L for crocetin vs. 2.0–5.5 mmol/L for crocin). This suggests that structural differences account for the different efficacies between them. Fluorescence-activated cell sorting (FACS) analysis showed that crocetin induced a significant level of cellular reactive oxygen species (ROS) in HeLa cells, whereas crocin did not. This ROS induction supported the cytotoxicity of crocetin, but not of crocin. A significant activation of nuclear factor erythroid 2-related factor 2 (Nrf2) was observed in both HeLa cells treated with crocin and crocetin: a 3.0-fold increase by 1 mmol/L crocetin and a 1.6-fold increase by 0.8 mmol/L crocin compared to the control. Furthermore, both crocetin and crocin reduced the protein expression of lactate dehydrogenase A (LDHA), one of the targets for chemoprevention in cancer cells, by 34.2% and 10.5%, respectively, compared to the control in HeLa cells. These findings suggest that crocetin and crocin have different mechanisms for their observed cytotoxicity in cancer cell lines.
      PubDate: Wed, 29 Jan 2014 08:00:00 GMT
       
  • Cell differentiation along multiple pathways accompanied by changes in
           histone acetylation status
    • Authors: Soňa Legartová; Stanislav Kozubek, Michal Franek, Zbyněk Zdráhal, Gabriela Lochmanová, Nadine Martinet, Eva Bártová
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 85-93, e-First articles. Post-translational modification of histones is fundamental to the regulation of basic nuclear processes and subsequent cellular events, including differentiation. In this study, we analyzed acetylated forms of histones H2A, H2B, and H4 during induced differentiation in mouse (mESCs) and human (hESCs) embryonic stem cells and during induced enterocytic differentiation of colon cancer cells in vitro. Endoderm-like differentiation of mESCs induced by retinoic acid and enterocytic differentiation induced by histone deacetylase inhibitor sodium butyrate were accompanied by increased mono-, di-, and tri-acetylation of histone H2B and a pronounced increase in di- and tri-acetylation of histone H4. In enterocytes, mono-acetylation of histone H2A also increased and tetra-acetylation of histone H4 appeared only after induction of this differentiation pathway. During differentiation of hESCs, we observed increased mono-acetylation and decreased tri-acetylation of H2B. Mono-, di-, and tri-acetylation of H4 were reduced, manifested by a significant increase in nonacetylated H4 histones. Levels of acetylated histones increased during induced differentiation in mESCs and during histone deacetylase (HDAC) inhibitor-induced enterocytic differentiation, whereas differentiation of human ESCs was associated with reduced acetylation of histones H2B and H4.
      PubDate: Fri, 17 Jan 2014 08:00:00 GMT
       
  • M3 muscarinic acetylcholine receptor dysfunction inhibits Rac1 activity
           and disrupts VE-cadherin/β-catenin and actin cytoskeleton interaction
           
    • Authors: Zhou-Yang Jiao; Jing Wu, Bing Wen, Wen-Zeng Zhao, Xin-Ling Du
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 137-144, e-First articles. The objective was to investigate whether M3 muscarinic acetylcholine receptor (mAChR) dysfunction disrupts the linkage between the vascular endothelial (VE)-cadherin in the adherens junctional complex and the actin-based cytoskeleton, increasing vascular permeability in atherosclerosis. Western blotting revealed that a selective M3 receptor antagonist, 4-diphenylacetoxy-N-methylpiperidine methiodide (4-DAMP), and M3 receptor siRNA decrease VE-cadherin and β-catenin in Triton X-100–insoluble fractions, indicating that M3 receptor inhibition weakens the linkage between the VE-cadherin/β-catenin complex and the actin cytoskeleton. Co-immunoprecipitation assays showed that M3 receptor inhibition reduces Rac1 activity and the association of IQ motif-containing GTPase-activating protein 1 (IQGAP1) with Ras-related C3 botulinum toxin substrate 1 (Rac1), while increasing the interaction between IQGAP1 and β-catenin. Using IQGAP1 siRNA, we found that IQGAP1 is required for stable interaction between VE-cadherin/β-catenin and the actin cytoskeleton in quiescent endothelial cells; IQGAP1 siRNA augments the M3 receptor inhibition-induced dissociation between them. Moreover, S-nitroso-N-acetylpenicillamine (SNAP), a nitric oxide (NO) donor, attenuates this disassociation and Rac1 activity inhibition. The M3 receptor facilitates interaction of the VE-cadherin–based adherens junctional complex and the actin-based cytoskeleton by maintaining Rac1 activity, which regulates the interaction between IQGAP1/Rac1 and IQGAP1/β-catenin, and may contribute to endothelial barrier function under physiological conditions.
      PubDate: Wed, 08 Jan 2014 08:00:00 GMT
       
  • Ectonucleotidase expression profile and activity in human cervical cancer
           cell lines
    • Authors: Aline Beckenkamp; Danielle Bertodo Santana, Alessandra Nejar Bruno, Luciane Noal Calil, Emerson André Casali, Juliano Domiraci Paccez, Luiz F. Zerbini, Guido Lenz, Márcia R. Wink, Andréia Buffon
      Abstract: Biochemistry and Cell Biology, Volume 0, Issue 0, Page 95-104, e-First articles. Cervical cancer is the third most frequent cancer in women worldwide. Adenine nucleotide signaling is modulated by the ectonucleotidases that act in sequence, forming an enzymatic cascade. Considering the relationship between the purinergic signaling and cancer, we studied the E-NTPDases, ecto-5′-nucleotidase, and E-NPPs in human cervical cancer cell lines and keratinocytes. We evaluated the expression profiles of these enzymes using RT-PCR and quantitative real-time PCR analysis. The activities of these enzymes were examined using ATP, ADP, AMP, and p-nitrophenyl-5′-thymidine monophosphate (p-Nph-5′-TMP) as substrate, in a colorimetric assay. The extracellular adenine nucleotide hydrolysis was estimated by HPLC analysis. The hydrolysis of all substrates exhibited a linear pattern and these activities were cation-dependent. An interesting difference in the degradation rate was observed between cervical cancer cell lines SiHa, HeLa, and C33A and normal imortalized keratinocytes, HaCaT cells. The mRNA of ecto-5′-nucleotidase, E-NTPDases 5 and 6 were detectable in all cell lines, and the dominant gene expressed was the Entpd 5 enzyme, in SiHa cell line (HPV16 positive). In accordance with this result, a higher hydrolysis activity for UDP and GDP nucleotides was observed in the supernatant of the SiHa cells. Both normal and cancer cells presented activity and mRNAs of members of the NPP family. Considering that these enzymes exert an important catalytic activity, controlling purinergic nucleotide concentrations in tumors, the presence of ectonucleotidases in cervical cancer cells can be important to regulate the levels of extracellular adenine nucleotides, limiting their effects.
      PubDate: Wed, 08 Jan 2014 08:00:00 GMT
       
 
 
JournalTOCs
School of Mathematical and Computer Sciences
Heriot-Watt University
Edinburgh, EH14 4AS, UK
Email: journaltocs@hw.ac.uk
Tel: +00 44 (0)131 4513762
Fax: +00 44 (0)131 4513327
 
About JournalTOCs
API
Help
News (blog, publications)
JournalTOCs on Twitter   JournalTOCs on Facebook

JournalTOCs © 2009-2014